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: Excessive alcohol consumption in adolescence disrupts neurodevelopment and promotes pain hypersensitivity. In rats; adolescent chronic intermittent ethanol (aCIE) exposure facilitates dopamine release in the middle NAc shell with an associated augmentation in pain sensitivity; an effect reversed by chemogenetic inhibition of dopamine. Multiple receptor systems; such as the kappa opioid receptor (KOR) inhibits dopamine release in a region-specific manner along the NAc rostro-caudal axis. Like dopamine; KORs in the NAc are involved in regulating alcohol use and pain sensitivity. Thus; here we examine the KOR-dopamine interaction in rostral and caudal NAc shell during protracted withdrawal and the associated impact on pain sensitivity.
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OpenAlex
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2024

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: Excessive alcohol consumption in adolescence disrupts neurodevelopment and promotes pain hypersensitivity. In rats; adolescent chronic intermittent ethanol (aCIE) exposure facilitates dopamine release in the middle NAc shell with an associated augmentation in pain sensitivity; an effect reversed by chemogenetic inhibition of dopamine. Multiple receptor systems; such as the kappa opioid receptor (KOR) inhibits dopamine release in a region-specific manner along the NAc rostro-caudal axis. Like dopamine; KORs in the NAc are involved in regulating alcohol use and pain sensitivity. Thus; here we examine the KOR-dopamine interaction in rostral and caudal NAc shell during protracted withdrawal and the associated impact on pain sensitivity. (2024). Poster Abstract. https://openalex.org/W4399297568

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URL permanente de la ficha: https://brain.psiconautica.org/documentos/d-a077821a741eebebd6ce470165a85570/